autoclaved food (PMI Nutrition International LLC)
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PMI Nutrition International LLC
autoclaved food
Autoclaved Food, supplied by PMI Nutrition International LLC, used in various techniques. Bioz Stars score: 96/100, based on 693 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/autoclaved+food/PicoLab+Mouse+Diet+20+5058/pmc12752765-355-0-2
Average 96 stars, based on 693 article reviews
Autoclaved Food, supplied by PMI Nutrition International LLC, used in various techniques. Bioz Stars score: 96/100, based on 693 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/autoclaved+food/PicoLab+Mouse+Diet+20+5058/pmc12752765-355-0-2
Average 96 stars, based on 693 article reviews
autoclaved food - by Bioz Stars,
2026-09
96/100 stars
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other:Article Title: Growth Factor-Independent mTORC1 Signaling Promotes Primary Cilia Length via Suppression of Autophagy Article Snippet: Article Title: AKT-mediated phosphorylation of TSC2 controls stimulus- and tissue-specific mTORC1 signaling and organ growth. Article Snippet: Short article AKT-mediated phosphorylation of TSC2 controls stimulus- and tissue-specific mTORC1 signaling and organ growth Graphical abstract Highlights • A phospho-mutant mouse (TSC2-5A) disconnecting Akt from TSC2 and mTORC1 is described • TSC2-5A mice develop normally but show an organ-specific reduction in weight • TSC2-5A mice have smaller brains and neurons, with reduced mTORC1 signaling • TSC2-5A mice have reduced muscle mass and feedinginduced muscle protein synthesis Authors Yann Cormerais, Samuel C. Lapp, Krystle C. Kalafut, ..., Suchithra Menon, Vanessa Byles, Brendan D. Manning Correspondence bmanning@hsph.harvard.edu In brief Cormerais et al. describe the TSC2-5A mouse model lacking the five phosphorylation sites on TSC2 through which AKT activates mTORC1.. The TSC2- 5A mice are developmentally normal but show reduced body- and organ-specific weight associated with cell-intrinsic defects in growth factor-stimulated mTORC1 signaling, cell growth, and protein synthesis.. Cormerais et al., 2025, Developmental Cell 60, 1–14 December 15, 2025 © 2025 Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies. https://doi.org/10.1016/j.devcel.2025.05.008 ll Article Title: AKT-mediated phosphorylation of TSC2 controls stimulus- and tissue-specific mTORC1 signaling and organ growth Article Snippet: Article Title: Adipocyte-specific deletion of Dbc1 does not recapitulate healthy obesity phenotype but suggests regulation of inflammation signaling Article Snippet: Article Title: Growth factor-independent mTORC1 signaling promotes primary cilia length via suppression of autophagy Article Snippet: Article Title: Regulate gut microbiota to treat neurodegenerative disorders Article Snippet: All animals, irrespective of colonization status, received autoclaved food ( Article Title: Adipocyte-specific deletion of Dbc1 does not recapitulate healthy obesity phenotype but suggests regulation of inflammation signaling. Article Snippet: Article Title: Adipocyte-specific deletion of Dbc1 does not recapitulate healthy obesity phenotype but suggests regulation of inflammation signaling Article Snippet: |